atical model, a unified map of not only the prevailing genes present in late Alzheimer's but the various pathways that they trigger. One such gene, TYROBP, not typically connected to Alzheimer's but linked to brain inflammation, was shown to interact with TREM2, a gene recently connected to Alzheimer's

atical model, a unified map of not only the prevailing genes present in late Alzheimer's but the various pathways that they trigger. One such gene, TYROBP, not typically connected to Alzheimer's but linked to brain inflammation, was shown to interact with TREM2, a gene recently connected to Alzheimer's

atical model, a unified map of not only the prevailing genes present in late Alzheimer's but the various pathways that they trigger. One such gene, TYROBP, not typically connected to Alzheimer's but linked to brain inflammation, was shown to interact with TREM2, a gene recently connected to Alzheimer's

a co-author of the study and Associate Professor of Genetics and Genomic Sciences at Mount Sinai. "We can now evaluate drugs that impact the TFtEM2- TYROBP pathway as potential therapies... and design more specific compounds that target these key steps precisely, in contrast to existing anti-inflammator