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otrusions, whose possible relationship to any functional asymmetries remains unclear. Regional hemispheric asym- metries both in cortical thickness (Luders et al., 2006) and volume (Good et al., 2001), both in gray and white matter (Penhune et al., 1996; Takao et al., 2011) have been reported. Any mor
r and the left hemisphere to cognitive familiarity and routinization. Since this functional asymmetry was first proposed (Goldberg and Costa, 1981; Goldberg et al., 1994a), it has found support with various neuroimaging tech- niques, including PET (Gold et al., 1996; Shadmehr and Holcomb, 1997), fMR1 (Henson e
otrusions, whose possible relationship to any functional asymmetries remains unclear. Regional hemispheric asym- metries both in cortical thickness (Luders et al., 2006) and volume (Good et al., 2001), both in gray and white matter (Penhune et al., 1996; Takao et al., 2011) have been reported. Any mor
r and the left hemisphere to cognitive familiarity and routinization. Since this functional asymmetry was first proposed (Goldberg and Costa. 1981; Goldberg et al., 1994a), it has found support with various neuroimaging tech- niques, including PET (Gold et al., 1996; Shadmehr and Holcomb, 1997), fMRI (Henson e
rized by shared morphometric properties also in the normal brain: both are larger in the left than in the right hemisphere (Goldberg et al., 2013; Luders et al., 2006). The fact that these two structures are characterized by the same direc- tion of lateralization both in the healthy brain and in the
he right hemi- sphere) versus exercise of established routinized behaviours (the left hemisphere) (Goldberg & Costa, 1981; Goldberg & Podell, 1995; Goldberg et al., 1994b). Within this general framework, the lateral- ized nature of the effects of frontal-lobe damage in humans has been demonstrated. Damage to t
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PERSONNew York University School of Medicine
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